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Items: 8

1.

Extended self-renewal and accelerated reprogramming in the absence of Kdm5b [ChIP-Seq]

(Submitter supplied) ES cell pluripotency is thought to be regulated in part by H3K4 methylation. However, it is unclear how H3K4 demethylation contributes to ES cell function and participates in iPS cell reprogramming. Here, we show that KDM5B, which demethylates H3K4, is important for ES cell differentiation, and presents a barrier to the reprogramming process. Depletion of Kdm5b leads to an extension in the self-renewal of ES cells in the absence of LIF. more...
Organism:
Mus musculus
Type:
Genome binding/occupancy profiling by high throughput sequencing
Platform:
GPL13112
6 Samples
Download data: BED, BEDGRAPH
Series
Accession:
GSE47123
ID:
200047123
2.

Illumina HiSeq 2000 (Mus musculus)

Platform
Accession:
GPL13112
ID:
100013112
3.

shKdm5b_4TF-MEF_Input_48h

Organism:
Mus musculus
Source name:
shKdm5b 4TF-MEFs
Platform:
GPL13112
Series:
GSE46893 GSE47123
Download data: BED, BEDGRAPH
Sample
Accession:
GSM1145402
ID:
301145402
4.

shLuc_4TF-MEF_Input_48h

Organism:
Mus musculus
Source name:
shLuc 4TF-MEFs
Platform:
GPL13112
Series:
GSE46893 GSE47123
Download data: BED, BEDGRAPH
Sample
Accession:
GSM1145401
ID:
301145401
5.

shKdm5b_4TF-MEF_H3K4me2_48h

Organism:
Mus musculus
Source name:
shKdm5b 4TF-MEFs
Platform:
GPL13112
Series:
GSE46893 GSE47123
Download data: BED, BEDGRAPH
Sample
Accession:
GSM1145400
ID:
301145400
6.

shKdm5b_4TF-MEF_H3K4me3_48h

Organism:
Mus musculus
Source name:
shKdm5b 4TF-MEFs
Platform:
GPL13112
Series:
GSE46893 GSE47123
Download data: BED, BEDGRAPH
Sample
Accession:
GSM1145399
ID:
301145399
7.

shLuc_4TF-MEF_H3K4me2_48h

Organism:
Mus musculus
Source name:
shLuc 4TF-MEFs
Platform:
GPL13112
Series:
GSE46893 GSE47123
Download data: BED, BEDGRAPH
Sample
Accession:
GSM1145398
ID:
301145398
8.

shLuc_4TF-MEF_H3K4me3_48h

Organism:
Mus musculus
Source name:
shLuc 4TF-MEFs
Platform:
GPL13112
Series:
GSE46893 GSE47123
Download data: BED, BEDGRAPH
Sample
Accession:
GSM1145397
ID:
301145397
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Supplemental Content

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