Loss of Bap1 leads to an increased global methylat... (ID 522474) - BioProject

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Accession: PRJNA522474 ID: 522474

Loss of Bap1 leads to an increased global methylation and decrease in H3K27AC enhancer marks around key lineage commitment genes in Xenopus laevis embryos [ChIP-seq] (African clawed frog)

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This project is a component of the Loss of Bap1 in Xenopus laevis embryos

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1 additional project is a component of the Loss of Bap1 in Xenopus laevis embryos.

294 additional projects are related by organism.

We performed morpholino-mediated knockdown of Bap1 protein expression in Xenopus laevis developing embryos, and analyzed inhibiting and activating histone marks . More...

Accession	PRJNA522474; GEO: GSE126598
Data Type	Epigenomics
Scope	Multiisolate
Organism	Xenopus laevis[Taxonomy ID: 8355] Eukaryota; Metazoa; Chordata; Craniata; Vertebrata; Euteleostomi; Amphibia; Batrachia; Anura; Pipoidea; Pipidae; Xenopodinae; Xenopus; Xenopus; Xenopus laevis
Publications	Kuznetsov JN et al., "BAP1 regulates epigenetic switch from pluripotency to differentiation in developmental lineages giving rise to BAP1-mutant cancers.", Sci Adv, 2019 Sep;5(9):eaax1738
Submission	Registration date: 14-Feb-2019 Harbour, Ophthalmology, University of Miami Miller School of Medicine
Relevance	Model Organism

Project Data:

Resource Name	Number of Links
Sequence data
SRA Experiments	20
Publications
PubMed	1
PMC	1
Other datasets
BioSample	20
GEO DataSets	1

GEO Data Details

Parameter	Value
Data volume, Supplementary Mbytes	2229

SRA Data Details

Parameter	Value
Data volume, Gbases	49
Data volume, Mbytes	19097

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Loss of Bap1 leads to an increased global methylation and decrease in H3K27AC enhancer marks around key lineage commitment genes in Xenopus laevis embryos [ChIP-seq] (African clawed frog)

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