This SuperSeries is composed of the SubSeries listed below.
Overall design: Refer to individual Series
| Accession | PRJNA522472; GEO: GSE126599 |
| Type | Umbrella project |
| Publications | Kuznetsov JN et al., "BAP1 regulates epigenetic switch from pluripotency to differentiation in developmental lineages giving rise to BAP1-mutant cancers.", Sci Adv, 2019 Sep;5(9):eaax1738 |
| Submission | Registration date: 14-Feb-2019
Harbour, Ophthalmology, University of Miami Miller School of Medicine |
| Relevance | Superseries |
Project Data:
| Resource Name | Number
of Links |
|---|
| Sequence data |
| SRA Experiments | 29 |
| Publications |
| PubMed | 1 |
| PMC | 1 |
| Other datasets |
| BioSample | 29 |
| GEO DataSets | 3 |
Loss of Bap1 in Xenopus laevis embryos encompasses the following 2 sub-projects:
| Project Type | Number of Projects |
| Epigenomics | 1 |
BioProject
accession | Organism | Title |
|---|
| PRJNA522474 | Xenopus laevis | Loss of Bap1 leads to an increased global methylation and decrease in H3K27AC enhancer marks around key lineage commitment genes in Xenopus laevis embryos [ChIP-seq] (Harbour, Ophthalmology, University...) |
|
| Transcriptome or Gene expression | 1 |
BioProject
accession | Organism | Title |
|---|
| PRJNA522475 | Xenopus laevis | Bap1 loss leads to decreased expression of lineage specific commitment genes, and increased expression of pluripotency genes [RNA-seq] (Harbour, Ophthalmology, University...) |
|