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Organizing biological data
PI3Kα inhibition blocks osteochondroprogenitor specification and the hyper-inflammatory response to prevent heterotopic ossification
Palovarotene action against heterotopic ossification includes the inhibition of local participating activin-A expressing cell populations
Transcriptomic differences underlying the Activin-A induced large osteoclast formation in both healthy control and Fibrodysplasia Ossificans Progressiva osteoclasts
Palovarotene action against heterotopic ossification includes the inhibition of local participating activin A-expressing cell populations
Oxidative phosphorylation is a pivotal therapeutic target of fibrodysplasia ossificans progressiva
RNA data of human FOP patients-derived iPSCs treated with ACVR1 R206H mutant-targeting AAVs
RNA-sequencing of forward programmed hepatocytes and fetal liver samples
Recapitulation of the pro-inflammatory signature of monocytes with ACVR1 mutation using FOP patient-derived iPSCs
Listeria reduction using a bacteriophage coctail
A bacteriophage cocktail significantly reduces Listeria monocytogenes without deleterious impact on the commensal gut microbiota under simulated gastro-intestinal conditions, and provides strong protective effect against adhesion and invasion in Caco-2 cells
Comparative transcriptomic analysis of races 1, 2 5 and 6 of Fusarium oxysporum f.sp. pisi in a susceptible pea host identifies differential pathogenicity profiles
Fusarium oxysporum f. sp. pisi
Fusarium oxysporum f. sp. pisi Genome sequencing and assembly
Control and FOP Human iPSC derived myogenic cell single cell RNA sequencing
Wisp inhibits chondrogenic differentiation in heterotopic ossification
Global gene expression profiling of FOP-iMSCs after chondrogenic differentiation with ERBB2 inhibitors
Homo sapiens
Global gene expression profiling of FOP- or resFOP-iMSCs after chondrogenic differentiation
Immunological Imbalance in Fibrodysplasia Ossificans Progressiva Revealed by PBMC Transcriptome Analysis
Concurrent progress of reprogramming and gene-correction to overcome limitation of gene therapy of ALK2 mutant iPSCs
Global gene expression profiling of FOP- or resFOP-iMSCs treated by several ligands
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