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Year Number of Results
1990 1
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2008 2
2011 1
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2023 1
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Bisphosphonate therapy for osteogenesis imperfecta.
Dwan K, Phillipi CA, Steiner RD, Basel D. Dwan K, et al. Cochrane Database Syst Rev. 2016 Oct 19;10(10):CD005088. doi: 10.1002/14651858.CD005088.pub4. Cochrane Database Syst Rev. 2016. PMID: 27760454 Free PMC article. Review.
BACKGROUND: Osteogenesis imperfecta is caused by a genetic defect resulting in an abnormal type I collagen bone matrix which typically results in multiple fractures with little or no trauma. Bisphosphonates are used in an attempt to increase bone mineral dens …
BACKGROUND: Osteogenesis imperfecta is caused by a genetic defect resulting in an abnormal type I collagen bone matrix …
The immunomodulatory role of matrix metalloproteinases in colitis-associated cancer.
He L, Kang Q, Chan KI, Zhang Y, Zhong Z, Tan W. He L, et al. Front Immunol. 2023 Jan 19;13:1093990. doi: 10.3389/fimmu.2022.1093990. eCollection 2022. Front Immunol. 2023. PMID: 36776395 Free PMC article. Review.
They are involved in diverse pathophysiological processes, such as tumor invasion and metastasis, cardiovascular diseases, arthritis, periodontal disease, osteogenesis imperfecta, and diseases of the central nervous system. ...Key MMPs that interfere with pathologic …
They are involved in diverse pathophysiological processes, such as tumor invasion and metastasis, cardiovascular diseases, arthritis, period …
Mortality and morbidity in patients with osteogenesis imperfecta in Denmark.
Folkestad L. Folkestad L. Dan Med J. 2018 Apr;65(4):B5454. Dan Med J. 2018. PMID: 29619932 Free article. Review.
Ostegenesis imperfecta (OI) is a hereditary disease of the connective tissue caused by mutations to, mainly, the genes that are involved in the biosynthesis of collagen type 1. ...The study included 39 patients with OI type I, and 39 healthy age and gender ma …
Ostegenesis imperfecta (OI) is a hereditary disease of the connective tissue caused by mutations to, mainly, the genes that are invol …
Severe cases of osteogenesis imperfecta type VIII due to a homozygous mutation in P3H1 (LEPRE1) and review of the literature.
Bala MM, Bala KA. Bala MM, et al. Adv Clin Exp Med. 2021 Dec;30(12):1233-1238. doi: 10.17219/acem/141367. Adv Clin Exp Med. 2021. PMID: 34637196 Free article. Review.
BACKGROUND: Osteogenesis imperfecta (OI) is a genetic disorder that causes skeletal fragility, multiple fractures and several extraskeletal disorders. Most cases of OI are caused by mutations in COL1A1/A2. Osteogenesis imperfecta type VIII typic …
BACKGROUND: Osteogenesis imperfecta (OI) is a genetic disorder that causes skeletal fragility, multiple fractures and several …
New perspectives on osteogenesis imperfecta.
Forlino A, Cabral WA, Barnes AM, Marini JC. Forlino A, et al. Nat Rev Endocrinol. 2011 Jun 14;7(9):540-57. doi: 10.1038/nrendo.2011.81. Nat Rev Endocrinol. 2011. PMID: 21670757 Free PMC article. Review.
A new paradigm has emerged for osteogenesis imperfecta as a collagen-related disorder. The more prevalent autosomal dominant forms of osteogenesis imperfecta are caused by primary defects in type I collagen, whereas autosomal recessive forms are …
A new paradigm has emerged for osteogenesis imperfecta as a collagen-related disorder. The more prevalent autosomal dominant f …
Deciphering the Relevance of Bone ECM Signaling.
Alcorta-Sevillano N, Macías I, Infante A, Rodríguez CI. Alcorta-Sevillano N, et al. Cells. 2020 Dec 7;9(12):2630. doi: 10.3390/cells9122630. Cells. 2020. PMID: 33297501 Free PMC article. Review.
The main inorganic components of the ECM are calcium-deficient apatite and trace elements, while the organic ECM consists of collagen type I and noncollagenous proteins. Bone ECM dynamically interacts with osteoblasts and osteoclasts to regulate the formation of new bone d …
The main inorganic components of the ECM are calcium-deficient apatite and trace elements, while the organic ECM consists of collagen typ …
Bisphosphonate therapy for osteogenesis imperfecta.
Dwan K, Phillipi CA, Steiner RD, Basel D. Dwan K, et al. Cochrane Database Syst Rev. 2014 Jul 23;(7):CD005088. doi: 10.1002/14651858.CD005088.pub3. Cochrane Database Syst Rev. 2014. PMID: 25054949 Updated. Review.
BACKGROUND: Osteogenesis imperfecta is caused by a genetic defect resulting in an abnormal type I collagen bone matrix which typically results in multiple fractures with little or no trauma. Bisphosphonates are used in an attempt to increase bone mineral dens …
BACKGROUND: Osteogenesis imperfecta is caused by a genetic defect resulting in an abnormal type I collagen bone matrix …
Bisphosphonate therapy for osteogenesis imperfecta.
Phillipi CA, Remmington T, Steiner RD. Phillipi CA, et al. Cochrane Database Syst Rev. 2008 Oct 8;(4):CD005088. doi: 10.1002/14651858.CD005088.pub2. Cochrane Database Syst Rev. 2008. PMID: 18843680 Updated. Review.
BACKGROUND: In osteogenesis imperfecta (OI) a genetic defect in type I collagen results in multiple fractures with little or no trauma. ...For spine BMD, no significant difference was noted in the aggregated data from two trials, MD 9.96 (95%CI -2.51 t …
BACKGROUND: In osteogenesis imperfecta (OI) a genetic defect in type I collagen results in multiple fractures with litt …
Stem cell transplantation before birth - a realistic option for treatment of osteogenesis imperfecta?
Westgren M, Götherström C. Westgren M, et al. Prenat Diagn. 2015 Sep;35(9):827-32. doi: 10.1002/pd.4611. Epub 2015 Jun 3. Prenat Diagn. 2015. PMID: 25962526 Review.
Osteogenesis imperfecta (OI) is characterized by severe bone deformities, growth retardation and bones that break easily, often from little or no apparent cause. OI is a genetic disorder primarily with defective type I collagen with a wide spectrum of clinica …
Osteogenesis imperfecta (OI) is characterized by severe bone deformities, growth retardation and bones that break easily, ofte …
Prolidase-dependent regulation of collagen biosynthesis.
Surazynski A, Miltyk W, Palka J, Phang JM. Surazynski A, et al. Amino Acids. 2008 Nov;35(4):731-8. doi: 10.1007/s00726-008-0051-8. Epub 2008 Mar 5. Amino Acids. 2008. PMID: 18320291 Review.
Prolidase [EC.3.4.13.9] is a cytosolic imidodipeptidase, which specifically splits imidodipeptides with C-terminal proline or hydroxyproline. ...It is of great importance during wound healing, inflammation, aging, tissue fibrosis and possibly skeletal abnormalities seen in …
Prolidase [EC.3.4.13.9] is a cytosolic imidodipeptidase, which specifically splits imidodipeptides with C-terminal proline or hydroxy …
16 results