Are the mechanisms for NO-dependent vascular remodeling different from vasorelaxation in vivo?

Arterioscler Thromb Vasc Biol. 2008 Jul;28(7):1207-8. doi: 10.1161/ATVBAHA.108.167403.
No abstract available

Publication types

  • Comment
  • Editorial

MeSH terms

  • Animals
  • Carotid Artery Injuries / metabolism*
  • Carotid Artery Injuries / pathology
  • Carotid Artery Injuries / physiopathology
  • Carotid Stenosis / metabolism*
  • Carotid Stenosis / pathology
  • Carotid Stenosis / physiopathology
  • Cyclic GMP / metabolism
  • Cyclic GMP-Dependent Protein Kinase Type I
  • Cyclic GMP-Dependent Protein Kinases / genetics
  • Cyclic GMP-Dependent Protein Kinases / metabolism
  • Cyclic Nucleotide Phosphodiesterases, Type 5 / metabolism
  • Disease Models, Animal
  • Mice
  • Mice, Knockout
  • Muscle, Smooth, Vascular / enzymology*
  • Muscle, Smooth, Vascular / pathology
  • Muscle, Smooth, Vascular / physiopathology
  • Nitric Oxide / metabolism*
  • Phosphodiesterase 5 Inhibitors
  • Phosphodiesterase Inhibitors / pharmacology
  • Piperazines / pharmacology
  • Purines / pharmacology
  • Recurrence
  • Signal Transduction* / drug effects
  • Sildenafil Citrate
  • Sulfones / pharmacology
  • Vasodilation*

Substances

  • Phosphodiesterase 5 Inhibitors
  • Phosphodiesterase Inhibitors
  • Piperazines
  • Purines
  • Sulfones
  • Nitric Oxide
  • Sildenafil Citrate
  • Cyclic GMP-Dependent Protein Kinase Type I
  • Cyclic GMP-Dependent Protein Kinases
  • Prkg1 protein, mouse
  • Cyclic Nucleotide Phosphodiesterases, Type 5
  • Cyclic GMP