Functional role of ICAM-3 polymorphism in genetic susceptibility to SARS infection

Hong Kong Med J. 2009 Oct:15 Suppl 6:26-9.

Authors

U S Khoo¹, K Y Chan, J C Y Ching, V S Chan, Y C Ip, L Yam, C M Chu, S T Lai, K M So, T Y Wong, P H Chung, P Tam, S P Yip, P Sham, G M Leung, C L Lin, J S M Peiris

Affiliation

¹ Department of Pathology, Li Ka Shing Faculty of Medicine, The University of Hong Kong, Hong Kong, China. uskhoo@pathology.hku.hk

PMID: 19801714

No abstract available

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Adolescent
Adult
Aged
Aged, 80 and over
Antigens, CD / genetics*
Cell Adhesion Molecules / genetics*
Cell Adhesion Molecules / physiology
Cells, Cultured
Child
Child, Preschool
Female
Genetic Association Studies
Genetic Predisposition to Disease*
Humans
L-Lactate Dehydrogenase / blood
Lectins, C-Type / genetics
Lectins, C-Type / physiology
Leukocyte Count
Male
Middle Aged
Polymorphism, Single Nucleotide*
Receptors, Cell Surface / genetics
Receptors, Cell Surface / physiology
Severe Acute Respiratory Syndrome / blood
Severe Acute Respiratory Syndrome / genetics*

Substances

Antigens, CD
Cell Adhesion Molecules
DC-specific ICAM-3 grabbing nonintegrin
ICAM3 protein, human
Lectins, C-Type
Receptors, Cell Surface
L-Lactate Dehydrogenase