SOCS1 and SHP1 hypermethylation in mantle cell lymphoma and follicular lymphoma: implications for epigenetic activation of the Jak/STAT pathway

Leukemia. 2004 Feb;18(2):356-8. doi: 10.1038/sj.leu.2403216.
No abstract available

Publication types

  • Letter

MeSH terms

  • Carrier Proteins / genetics*
  • Carrier Proteins / physiology
  • DNA Methylation*
  • DNA-Binding Proteins / metabolism
  • Epigenesis, Genetic
  • Humans
  • Intracellular Signaling Peptides and Proteins*
  • Janus Kinase 1
  • Lymphoma, Follicular / enzymology
  • Lymphoma, Follicular / etiology
  • Lymphoma, Follicular / genetics*
  • Lymphoma, Mantle-Cell / enzymology
  • Lymphoma, Mantle-Cell / etiology
  • Lymphoma, Mantle-Cell / genetics*
  • Protein Tyrosine Phosphatase, Non-Receptor Type 6
  • Protein Tyrosine Phosphatases / genetics*
  • Protein Tyrosine Phosphatases / physiology
  • Protein-Tyrosine Kinases / metabolism
  • Repressor Proteins / genetics*
  • Repressor Proteins / physiology
  • STAT1 Transcription Factor
  • Suppressor of Cytokine Signaling 1 Protein
  • Suppressor of Cytokine Signaling Proteins
  • Trans-Activators / metabolism

Substances

  • Carrier Proteins
  • DNA-Binding Proteins
  • Intracellular Signaling Peptides and Proteins
  • Repressor Proteins
  • SOCS1 protein, human
  • STAT1 Transcription Factor
  • STAT1 protein, human
  • Suppressor of Cytokine Signaling 1 Protein
  • Suppressor of Cytokine Signaling Proteins
  • Trans-Activators
  • Protein-Tyrosine Kinases
  • JAK1 protein, human
  • Janus Kinase 1
  • PTPN6 protein, human
  • Protein Tyrosine Phosphatase, Non-Receptor Type 6
  • Protein Tyrosine Phosphatases